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Holland. The experts verified that SP cells are highly tumorigenic, be capable of self-renew, and so are resistant to chemotherapy. These total outcomes verified that ABCG2 activity, although not alone oncogenic, is normally a marker for glioma stem-like cells. Further, the experts identified an in depth molecular system that modulates the experience of ABCG2 and enhances the power of tumor stem-like cells to expel medicines. Significantly, Dr. Holland’s group also discovered that the chemotherapeutic medication temozolomide, the typical treatment for gliomas, improved the amount of glioma cells with stem-like characteristics. The experts speculated that because temozolomide isn’t an ABCG2 substrate, the upsurge in the SP fraction most likely resulted from enrichment of cells with stem-like properties.